Bisabolangelone inhibits dendritic cell functions by blocking MAPK and NF-kappa B signaling

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dc.contributor.authorH S Kim-
dc.contributor.authorY J Lee-
dc.contributor.authorH K Lee-
dc.contributor.authorJ S Kim-
dc.contributor.authorY Park-
dc.contributor.authorJong Soon Kang-
dc.contributor.authorB Y Hwang-
dc.contributor.authorJ T Hong-
dc.contributor.authorY Kim-
dc.contributor.authorS B Han-
dc.date.accessioned2017-04-19T09:46:12Z-
dc.date.available2017-04-19T09:46:12Z-
dc.date.issued2013-
dc.identifier.issn0278-6915-
dc.identifier.uri10.1016/j.fct.2013.05.013ko
dc.identifier.urihttps://oak.kribb.re.kr/handle/201005/11630-
dc.description.abstractBisabolangelone (BISA), isolated from the roots of Angelica koreana, has many pharmacological activities, such as anti-tumor, anti-microbial, antioxidant, and anti-inflammatory activities. In this study, we investigated the anti-inflammatory mechanisms of BISA in dendritic cells (DCs), which play an essential role in innate and adaptive immune responses. BISA attenuated the production of pro-inflammatory cytokines including interleukin (IL)-12, IL-1β, and tumor necrosis factor-alpha (TNF-α), migration to macrophage inflammatory protein-3 beta, and allo-T cell activating ability of DCs. In addition, BISA affected endocytosis of DCs. Molecular studies showed that BISA suppressed MAPK phosphorylation and nuclear translocation of NF-κB p50/p65. Taken together, our data suggest that BISA inhibited DC functions by blocking MAPK and NF-κB signaling.-
dc.publisherElsevier-
dc.titleBisabolangelone inhibits dendritic cell functions by blocking MAPK and NF-kappa B signaling-
dc.title.alternativeBisabolangelone inhibits dendritic cell functions by blocking MAPK and NF-kappa B signaling-
dc.typeArticle-
dc.citation.titleFood and Chemical Toxicology-
dc.citation.number0-
dc.citation.endPage33-
dc.citation.startPage26-
dc.citation.volume59-
dc.contributor.affiliatedAuthorJong Soon Kang-
dc.contributor.alternativeName김형숙-
dc.contributor.alternativeName이영준-
dc.contributor.alternativeName이홍경-
dc.contributor.alternativeName김지성-
dc.contributor.alternativeName박윤수-
dc.contributor.alternativeName강종순-
dc.contributor.alternativeName황방연-
dc.contributor.alternativeName홍진태-
dc.contributor.alternativeName김영수-
dc.contributor.alternativeName한상배-
dc.identifier.bibliographicCitationFood and Chemical Toxicology, vol. 59, pp. 26-33-
dc.identifier.doi10.1016/j.fct.2013.05.013-
dc.subject.keywordBisabolangelone-
dc.subject.keywordDendritic cells-
dc.subject.keywordMAPKs-
dc.subject.keywordNF-κB-
dc.subject.localBisabolangelone-
dc.subject.localDendritic cell-
dc.subject.localDendritic cells (DC)-
dc.subject.localdendritic cells-
dc.subject.localDendritic cells (DCs)-
dc.subject.localDendritic cells-
dc.subject.localdendritic cells (DC)-
dc.subject.localdendritic cell-
dc.subject.localMAPK-
dc.subject.localMAPKs-
dc.subject.localNuclear factor-kappa B-
dc.subject.localnuclear factor κB-
dc.subject.localNf-κb-
dc.subject.localNF-kB-
dc.subject.localnuclear factor kappa B-
dc.subject.localNF-κB (nuclear factor kappa-B)-
dc.subject.localNF-kappaB-
dc.subject.localNuclear factor-κb-
dc.subject.localNF-κ B-
dc.subject.localNF-κB-
dc.subject.localNF-kappa B-
dc.subject.localNuclear factor κB (NF-κB)-
dc.subject.localNuclear factor κB-
dc.subject.localNFκB-
dc.subject.localNf-κB-
dc.subject.localNuclear factor-κB-
dc.subject.localnuclear factorκB-
dc.subject.localNuclear factor (NF)-κB-
dc.subject.localNuclear factor kappa B-
dc.subject.localnuclear factor-κB-
dc.subject.localNF-ΚB-
dc.subject.localNuclear factor-kappa B (NF-κB)-
dc.subject.localNuclear factor-kappaB-
dc.subject.localnuclear factor-kappaB-
dc.subject.localnuclear factor-kappaB (NF-κB)-
dc.subject.localNFkappaB-
dc.subject.localNuclear factor kappaB-
dc.description.journalClassY-
Appears in Collections:
Ochang Branch Institute > Division of National Bio-Infrastructure > Laboratory Animal Resource & Research Center > 1. Journal Articles
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