Critical roles of E2f3 in Growth and musculo-skeletal phenotype in mice

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dc.contributor.authorHae-Rim Kim-
dc.contributor.authorFaiz Ur Rahman-
dc.contributor.authorKwang Soo Kim-
dc.contributor.authorEun-Kyeung Kim-
dc.contributor.authorSang Mi Cho-
dc.contributor.authorKihoon Lee-
dc.contributor.authorOk Sung Moon-
dc.contributor.authorYoung Won Seo-
dc.contributor.authorWon Kee Yoon-
dc.contributor.authorYoung Suk Won-
dc.contributor.authorHoyoung Kang-
dc.contributor.authorHyoung-Chin Kim-
dc.contributor.authorKi Hoan Nam-
dc.date.accessioned2020-02-07T16:30:32Z-
dc.date.available2020-02-07T16:30:32Z-
dc.date.issued2019-
dc.identifier.issn1449-1907-
dc.identifier.uri10.7150/ijms.39068ko
dc.identifier.urihttps://oak.kribb.re.kr/handle/201005/19140-
dc.description.abstractE2F3, a member of the E2F family, plays a critical role in cell cycle and proliferation by targeting downstream, retinoblastoma (RB) a tumor suppressor family protein. The purpose of this study, was to investigate the role and function of E2F3 in vivo. We examined phenotypic abnormalities, by deletion of the E2f3 gene in mice. Complete ablation of the E2F3 was fully penetrant, in the pure C57BL/6N background. The E2f3+/ - mouse embryo developed normally without fatal disorder. However, they exhibited reduced body weight, growth retardation, skeletal imperfection, and poor grip strength ability. Findings suggest that E2F3 has a pivotal role in muscle and bone development, and affect normal mouse growth.-
dc.publisherIvyspring Int Publ-
dc.titleCritical roles of E2f3 in Growth and musculo-skeletal phenotype in mice-
dc.title.alternativeCritical roles of E2f3 in Growth and musculo-skeletal phenotype in mice-
dc.typeArticle-
dc.citation.titleInternational Journal of Medical Sciences-
dc.citation.number12-
dc.citation.endPage1563-
dc.citation.startPage1557-
dc.citation.volume16-
dc.contributor.affiliatedAuthorHae-Rim Kim-
dc.contributor.affiliatedAuthorKwang Soo Kim-
dc.contributor.affiliatedAuthorEun-Kyeung Kim-
dc.contributor.affiliatedAuthorSang Mi Cho-
dc.contributor.affiliatedAuthorKihoon Lee-
dc.contributor.affiliatedAuthorOk Sung Moon-
dc.contributor.affiliatedAuthorYoung Won Seo-
dc.contributor.affiliatedAuthorWon Kee Yoon-
dc.contributor.affiliatedAuthorYoung Suk Won-
dc.contributor.affiliatedAuthorHoyoung Kang-
dc.contributor.affiliatedAuthorHyoung-Chin Kim-
dc.contributor.affiliatedAuthorKi Hoan Nam-
dc.contributor.alternativeName김해림-
dc.contributor.alternativeNameRahman-
dc.contributor.alternativeName김광수-
dc.contributor.alternativeName김은경-
dc.contributor.alternativeName조상미-
dc.contributor.alternativeName이기훈-
dc.contributor.alternativeName문옥성-
dc.contributor.alternativeName서영원-
dc.contributor.alternativeName윤원기-
dc.contributor.alternativeName원영석-
dc.contributor.alternativeName강호영-
dc.contributor.alternativeName김형진-
dc.contributor.alternativeName남기환-
dc.identifier.bibliographicCitationInternational Journal of Medical Sciences, vol. 16, no. 12, pp. 1557-1563-
dc.identifier.doi10.7150/ijms.39068-
dc.subject.keywordDevelopment-
dc.subject.keywordE2f3+/- Knockout-
dc.subject.keywordGrowth retardation-
dc.subject.keywordMouse phenotype-
dc.subject.keywordskeletal imperfection-
dc.subject.localdevelopment-
dc.subject.localdeveoopment-
dc.subject.localDevelopment-
dc.subject.localE2f3+/- Knockout-
dc.subject.localgrowth retardation-
dc.subject.localGrowth retardation-
dc.subject.localMouse phenotype-
dc.subject.localskeletal imperfection-
dc.description.journalClassY-
Appears in Collections:
Ochang Branch Institute > Division of National Bio-Infrastructure > Laboratory Animal Resource & Research Center > 1. Journal Articles
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